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I volumi curati e tradotti da. Lettere a Wolfgang Kraus (1. Emil Cioran. Intervista con Christian Bussy, di Emil Cioran (La scuola di Pitagora editrice, Napoli, 2. Intervista con Philippe D. Dracodaidis (La scuola di Pitagora editrice, Napoli, 2. Ineffabile nostalgia. Lettere al fratello 1. I suoi articoli pubblicati includono. Prospettive Antimoderne, n. Il paradosso romeno Eliade, Cioran e la . La conversazione, a un certo punto, prese una piega insolita. Insomma, una delle tante meteore che attraversano la storia della musica pop, eroi d. Per quanto mi riguarda ho appreso molto di pi. Un popolo di vinti. Prima ancora di essere una questione individuale, il fallimento, almeno in certi Paesi che si trascinano nelle periferie della storia, assume i contorni funesti d. Scetticismo superficiale, passivit. Il termine chiave romeno . Cresce su un fondo di sofferenza e si espande, aerea, sopra la prostrazione di un popolo, estraneo alla felicit. Occorre pensare alla sua storia di sconforto, al cumulo di disgrazie, d. Il proprio Paese si trova quindi davanti a un bivio, a un. Se la Romania, dopo secoli di sordida esistenza, non irromper. Imponendo un ultimatum al Paese, egli chiarisce subito di non essere disposto, in caso d. Nel 1. 93. 7, traendo le conseguenze da quell. Salvo un fugace e tempestoso rientro tra il novembre del . Tra vaghi propositi di ricerca filosofica e incerte borse di studio, si ritrova in realt. Ogni volta che osservo quelle ombre umane, studenti romeni, polacchi, spagnoli o cinesi (hanno tutti la vocazione all. La vita non si realizza n. Iniziato alla scienza della caducit. Il prezzo pagato per la superba inutilit. Proprio in quelle petites chambres d. Pur essendo un esemplare tipicamente balcanico, lo si ritrova un po. L'inconveniente di essere nati PDF. Al culmine della disperazione; Sommario di decomposizione; La caduta nel tempo; Home; Disclaimer; DMCA. Sommario di decomposizione L'amore. Emil Cioran, Sommario di decomposizione. Scarica come PDF; Versione stampabile. Cioran Sommario Di Decomposizione Pdf MergeUn amore di Cioran, di Friedgard Thoma (Orecchio di Van Gogh, Falconara Marittima, 2010), L'agonia dell'Occidente. Sommario di decomposizione (Biblioteca Adelphi) Adelphi . Tutti i libri di Cioran Emil M. Chi sceglie questo libro legge anche: Benn Gottfried. Cioran ne azzarda una definizione: . Non si abbassa a lasciare un. Si adatta a vivere da parassita dell. Inchiodato ai blocchi di partenza, non prende parte alla gara della vita, ben sapendo che tutti i partecipanti, presto o tardi, inciamperanno rovinosamente in qualche ostacolo e che nessuno, veramente nessuno, taglier. 1993 Formato del file: Pdf Epub Mobi. Orgoglio supremo del fallito, rivincita estrema del perdente, la si chiami come si vuole, resta il fatto che le evidenze si schierano a ranghi serrati dalla sua parte. Ma, nel nonsenso universale, noi c. Tuttavia, quanti ne sono consapevoli? Hanno dei cammini da seguire, delle mete da raggiungere, dei bisogni da saziare. Non sanno gioire della propria incompiutezza, mentre la vita non si giustifica altrimenti che per l. Solo in quel momento, . E comprendere significa . Frutto di una congiuntura favorevole quanto imprevedibile di condizioni e accadimenti, la cosiddetta . Illudendolo di essere stato lui a pilotare il corso degli eventi verso il raggiungimento del risultato, il successo lo adula, lo lusinga, inoculandogli il veleno dell. Mostri di superficialit. In altre parole, diventer. Senza quella disciplina di fronte all? Quella saggezza funebre, vera e propria metafisica rurale che un tempo lo riempiva di vergogna, ora diventa un segno d. In particolare, rimane indelebile nella memoria di Cioran la figura d. Costui, dopo aver ereditato una fortuna da uno zio d. Ora quando ripenso a lui, persisto nel credere che egli fosse veramente qualcuno, che tra tutti gli abitanti del Paese lui solo avesse abbastanza levatura da sprecare la sua vita. Cioran non si scompose, anzi, fu visibilmente contrariato dall. Da Parigi, a stretto giro di posta, arriv. In aperto dissenso con l. Una vittima invincibile? Un recalcitrante divino, un eroe . Cioran, Transfiguration de la Roumanie, . I riferimenti bibliografici cioraniani privi del nome dell. Cioran, Transfiguration de la Roumanie, cit., p. Protagonista del poema . Sebbene avvertito per tempo da una sua agnellina (Miori. Cioran, Les secrets de l. Cioran, Transfiguration de la Roumanie, cit. Cioran, Profilul interior al c. Cioran, Transfiguration de la Roumanie, cit. Cioran, Fragments de Quartier latin, in . Gabriel Liiceanu, Itin. Cioran, Michalon, Paris 1. Cioran, Mihai Eminescu, in Exercices n. Altrove si descrive come . Cioran, Cahiers 1. Gallimard, Paris 1. Cioran, Fragments de Quartier latin, cit., p. 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Low Molecular Weight Heparins, Biological Drugs close to Non. The history and historical treatments of deep vein thrombosis. We also bring some perspective to historical. Use of novel oral anticoagulant agents in atrial fibrillation: current evidence and future. Discovery of anticoagulant drugs: a historical perspective. PE and deep vein thrombosis (DVT) are considered expressions of the same disease, termed as venous thromboembolism (VTE). In the present review, we describe and meta- analyze the efficacy and safety data available with the direct oral anticoagulants (DOAC; dabigatran, rivaroxaban, apixaban, edoxaban) in clinical trials testing these new compounds in the acute/long- term and extended therapy of VTE, providing subgroup analyses in patients with index PE. We analyzed ten studies in 3. A total of 1. 4,3. PE. In the acute/long- term treatment of VTE, the DOAC showed comparable efficacy in preventing recurrent VTE to standard treatment in patients with index PE (risk ratio . VTE recurrence depending on PE anatomical extension and presence/absence of right ventricular dysfunction was only reported in two trials, with results being consistent with those obtained in the overall study populations. In the single trial comparing extended therapy of VTE with DOAC versus warfarin, the point estimate for recurrent VTE tended to disfavor the DOAC in patients with index PE (RR: 2. CI: 0. 8. 3–5. 0. DVT (RR: 1. 1. 1; 9. CI: 0. 4. 9–2. 5. P for subgroup differences =0. In trials that compared DOAC versus placebo for extended therapy, the reduction in recurrent VTE was consistent in patients with PE (RR: 0. CI: 0. 0. 1–1. 8. DVT (RR: 0. 2. 5; 9. CI: 0. 1. 0–0. 6. P for subgroup differences =0. The DOAC were associated with a consistently lower risk of clinically relevant bleeding (CRB) than standard treatment of acute VTE and higher risk of CRB than placebo for extended therapy of VTE regardless of index event. In summary, the DOAC were as effective as, and safer than, standard treatment of (hemodynamically stable) PE. Their efficacy in preventing recurrent VTE seemed consistent regardless of anatomical extension of PE (extensive, intermediate, or limit) or presence/absence of right ventricular dysfunction although the data are limited. For extended therapy, the DOAC were more effective than placebo in preventing recurrent VTE but were associated with an increase in CRB regardless of index event. Keywords: anticoagulant, pulmonary embolism, dabigatran, apixaban, rivaroxaban, edoxaban. Introduction. Pulmonary embolism (PE) is a relatively common cardiovascular emergency. PE occurs when clots break off from deep vein walls (deep vein thrombosis . The broader term venous thromboembolism (VTE) refers to DVT, PE, or a combination of both, and has a reported annual incidence of 1–2 cases per 1,0. Western countries. PE and DVT are considered as expressions of the same disease, as in different studies, about 7. PE had DVT,2 and patients presenting with documented DVT have been shown to have silent PE or perfusion defects on ventilation/perfusion lung scan in 3. PE can be broadly classified as either high- risk PE (hemodynamically unstable: presence of shock or hypotension, defined as a systolic blood pressure < 9. Hg or a pressure drop of . Intermediate- risk PE (early mortality risk . Some clinical scores, such as the simplified PE severity index, may also be useful to identify low- risk PE patients who are potential candidates for outpatient treatment. Patients presenting with hemodynamic instability (high- risk PE) are usually treated with either thrombolytic therapy or pulmonary embolectomy followed by anticoagulant therapy, while most hemodynamically stable patients (non- high risk PE) can be treated with anticoagulation alone. The basic anticoagulant treatment strategy is similar for patients with PE or DVT, and generally includes a parenteral anticoagulant (eg, low- molecular- weight heparin (LMWH), unfractionated heparin, or fondaparinux) for at least 5 days and until the international normalized ratio (INR) is 2. K antagonist (VKA) (dose adjusted to keep an INR between 2. At this point, the decision to continue with anticoagulant treatment depends on the balance between the risk of VTE recurrences if anticoagulation is withdrawn and the risk of bleeding if it is continued. Patients with ongoing risk factors and patients with unprovoked VTE are often prescribed extended therapy, provided that the bleeding risk is not excessive. Despite LMWH or fondaparinux being effective and safe, they still require daily parenteral subcutaneous administration. In addition, they are mainly cleared through the kidneys, and their use in patients with severe renal insufficiency may be problematic. On the other hand, the narrow therapeutic window and variability in response of VKA implies that frequent anticoagulant monitoring (using the prothrombin time and its reporting as the INR) is necessary to avoid a subtherapeutic anticoagulation associated with an increased risk of thrombosis or an excessive anticoagulation that increases the risk of bleeding. Such monitoring is inconvenient for patients and medical staff, and costly for health care payers. In recent years, several direct oral anticoagulants (DOAC) have been developed for the treatment of VTE, among other indications. DOAC directly inhibit either thrombin or activated factor X (FXa). Among them, the oral direct thrombin inhibitor dabigatran etexilate (Pradaxa. Their pharmacology is well characterized (Table 1). They are given orally in fixed doses once or twice daily, and they lack validated and available antidotes. Table 1 Pharmacological characteristics of the old and new anticoagulants used for treatment of VTENotes: *Dalteparin (first approved LMWH in 1. We also searched regulatory agencies’ websites (European Medicines Agency, US Food and Drug Administration) and relevant conference proceedings related to anticoagulant therapy. Study selection criteria. We included randomized controlled trials comparing a DOAC (dabigatran, rivaroxaban, apixaban, and edoxaban) with the standard treatment for acute VTE or with warfarin or placebo for extended treatment of VTE. Data extraction and quality assessment. We collected outcome data on the overall trial populations as well as by index event at baseline: index PE (with or without concomitant DVT) or index DVT (without PE). Primary efficacy outcome was recurrent symptomatic VTE (ie, the composite of recurrent DVT, recurrent non- fatal PE, and VTE- related death) and primary safety outcome was clinically relevant bleeding (CRB) (ie, the composite of major and clinically relevant non- major bleeding). We also extracted data on clinical trials design (eg, superiority, non- inferiority, duration of treatment and follow- up, inclusion and exclusion criteria, and outcomes definitions), treatment characteristics (eg, dosage used in the experimental and control groups), and patients’ characteristics (age and sex; percentage of patients evaluable for efficacy and safety). Study quality was assessed using the Cochrane Collaboration’s tool for assessing risk of bias in randomized studies. Data synthesis and analysis. We carried out direct comparisons between the DOAC and standard treatment (initial/long- term therapy), placebo or warfarin (extended therapy) on an intention- to- treat basis. We included the randomized population in the overall analysis. Clinical outcomes were analyzed in subgroups of patients with index PE versus those with index DVT when disaggregated data were available. Subgroup analyses by index event (PE or DVT) were conducted in the assessable population for efficacy and safety, as reported in the original publications. A random effects meta- analysis was carried out by conventional methods, as described by Der. Simonian and Laird. Heterogeneity was assessed using the Cochran Q test. Higgins I2 test. 3. A Cochran’s Q P< 0. I2> 5. 0% were considered to show significant heterogeneity. Calculations were performed using Rev. Man software, version 5. The Nordic Cochrane Centre, Copenhagen, Denmark) and Stats. Direct software, version 2. Stats. Direct Ltd, Cheshire, UK). Results. Characteristics of studies and patients. Table 2 shows the characteristics of the studies and patients. We included ten studies in 3. A total of 1. 4,3. PE. Table 2 Clinical trials with DOAC in the treatment of VTENotes: *Extensive: clots in . Of them, 1. 1,6. 13 patients (4. PE. The four studies in the extended therapy of VTE3. Table 2). Of them, 2,7. PE. Mean age of patients was 5. Table 2). Ethnic group distribution was reported in seven trials. Across studies, the majority of patients included were Caucasians (7. Asians (2%–2. 1%) and Blacks or African Americans (1%–4%). Extensive PE was present in 2. PE (data only available from Oral Direct Factor Xa Inhibitor Rivaroxaban in Patients With Acute Symptomatic Pulmonary Embolism . Treatment durations ranged between 3 months and 1. VTE and between 6 months and 1. VTE (Table 2). All studies in the acute/long- term therapy of VTE and Secondary Prevention of Venous Thrombo Embolism (VTE) (RE- MEDY)4. VTE used a non- inferiority approach, while placebo- controlled studies in extended therapy of VTE were superiority trials (Table 2). In placebo- controlled trials, patients were recruited if there was clinical equipoise about the continuation or cessation of anticoagulant therapy. All studies but two (Oral Direct Factor Xa Inhibitor Rivaroxaban in Patients With Acute Symptomatic Deep Vein Thrombosis . Methods for assessment of recurrent VTE were consistent across studies. DVT diagnosis was established by compression ultrasonography or venography of leg veins, while non- fatal PE was diagnosed by ventilation–perfusion lung scanning, angiography, or spiral computed tomography of pulmonary arteries. Diagnosis of fatal PE was based on objective testing, autopsy, or death that could not be attributed to a documented cause and for which PE could not be ruled out. CRB definition was based on that used by the van Gogh trialists. Time in therapeutic range (TTR) ranged between 5. 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